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Anatomic Considerations This nerve arises from the lateral surface of the medulla by a series of small roots that lie just rostral to those of the vagus nerve The glossopharyngeal, vagus, and spinal accessory nerves leave the skull together through the jugular foramen and are then distributed peripherally The ninth nerve is mainly sensory, with cell bodies in the inferior, or petrosal, ganglion (the central processes of which end in the nucleus solitarius) and the small superior ganglion (the central bers of which enter the spinal trigeminal tract and nucleus) Within the nerve are afferent bers from baroreceptors in the wall of the carotid sinus and from chemoreceptors in the carotid body The baroreceptors are involved in the regulation of blood pressure, and chemoreceptors are responsible for the ventilatory responses to hypoxia The somatic efferent bers of the ninth nerve are derived from the nucleus ambiguus, and the visceral efferent (secretory) bers, from the inferior salivatory nucleus These bers contribute in a limited way to the motor innervation of the striated musculature of the pharynx (mainly of the stylopharyngeus, which elevates the pharynx), the parotid gland, and the glands in the pharyngeal mucosa A discussion of its role in swallowing is found on page 476) It is commonly stated that this nerve mediates sensory impulses from the faucial tonsils, posterior wall of the pharynx, and part of the soft palate as well as taste sensation from the posterior third of the tongue However, an isolated lesion of the ninth cranial nerve is a rarity, and the effects are not fully known In one personally observed case of bilateral surgical interruption of the ninth nerves, veri ed at autopsy, there had been no demonstrable loss of taste or other sensory or motor impairment This suggests that the tenth nerve may be responsible for these functions, at least in some individuals The role of the ninth nerve in the re ex control of blood pressure and ventilation has been alluded to earlier but referable clinical manifestations from damage of this cranial nerve are infrequent except perhaps for syncope as noted below One may occasionally observe a glossopharyngeal palsy in conjunction with vagus and accessory nerve involvement due to a tumor in the posterior fossa or an aneurysm of the vertebral artery The nerves are compressed as they pass through the jugular foramen Hoarseness due to vocal cord paralysis, some dif culty in swallowing, deviation of the soft palate to the sound side, anesthesia of the posterior wall of the pharynx, and weakness of the upper trapezius and sternomastoid muscles make up the clinical picture (see Table 47-1, jugular foramen syndrome) On leaving the skull, the ninth, tenth, and eleventh nerves lie adjacent to the internal carotid artery, where they can be damaged by a dissection of that vessel Glossopharyngeal Neuralgia (see also page 163) This disorder rst described by Weisenburg in 1910 resembles trigeminal neuralgia in many respects except that the unilateral stabbing pain is localized to one side of the root of the tongue and throat It is far less common than trigeminal neuralgia Sometimes the pain overlaps the vagal territory beneath the angle of the jaw and external auditory meatus It may be triggered by coughing, sneezing, swallowing, and pressure on the tragus of the ear Temporary blocking of the pain by anesthetizing the tonsillar fauces and posterior pharynx with 10% lidocaine spray is diagnostic Rarely, herpes zoster.

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may involve the glossopharyngeal nerve Fainting as a manifestation of vagoglossopharyngeal neuralgia is described on page 325 The same drugs that have been found helpful in the treatment of tic douloureux may be used to treat glossopharyngeal neuralgia, but their ef cacy has been dif cult to judge Regarding vascular compression of the nerve, Resnick and colleagues have reported the results of microvascular decompression of the ninth nerve in 40 patients; in 32 of these, relief of symptoms was complete and was sustained during an average follow-up of 4 years; 3 patients remained with permanent weakness of structures thought to be innervated by the ninth nerve A similar high rate of success has been achieved by others If syncope is associated with the pain, it can be expected to cease with abolition of the attacks of pain Syncope can also occur when the ninth nerve is involved by tumors of the parapharyngeal space; most of these are squamous cell carcinomas and both the ninth and tenth nerves are implicated Section of rootlets of the ninth nerve has reportedly reduced or abolished the episodes of fainting in these cases

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Anatomic Considerations This nerve has an extensive sensory and motor distribution and important autonomic functions It has two ganglia: the jugular, which contains the cell bodies of the somatic sensory nerves (innervating the skin in the concha of the ear), and the nodose, which contains the cell bodies of the afferent bers from the pharynx, larynx, trachea, esophagus, and thoracic and abdominal viscera The central processes of these two ganglia terminate in relation to the nucleus of the spinal trigeminal tract and the tractus solitarius, respectively The motor bers of the vagus are derived from two nuclei in the medulla the nucleus ambiguus and the dorsal motor nucleus The former supplies somatic motor bers to the striated muscles of the larynx, pharynx, and palate; the latter supplies visceral motor bers to the heart and other thoracic and abdominal organs The distribution of vagal bers is illustrated in Fig 47-4, and their participation in swallowing is described on page 476 Complete interruption of the intracranial portion of one vagus nerve results in a characteristic paralysis The soft palate droops on the ipsilateral side and does not rise in phonation The uvula deviates to the normal side on phonation, but this is an inconstant sign in disease There is loss of the gag re ex on the affected side and of the curtain movement of the lateral wall of the pharynx, whereby the faucial pillars move medially as the palate rises in saying ah The voice is hoarse, often nasal, and the vocal cord on the affected side lies immobile in a cadaveric position, ie, midway between abduction and adduction With partial lesions, movements of abduction are affected more than those of adduction (Semon s law) There may be a loss of sensation at the external auditory meatus and back of the pinna Usually no change in visceral function can be demonstrated If the pharyngeal branches of both vagi are affected, as in diphtheria, the voice has a nasal quality, and regurgitation of liquids through the nose occurs during the act of swallowing Diseases Affecting the Vagus Complete bilateral paralysis is said to be incompatible with life, and this is probably true if the nuclei are entirely destroyed in the medulla by poliomyelitis or some other disease However, in the cervical region, both vagi have been blocked with procaine in the treatment of intractable asthma without mishap Moreover, Johnson and Stern report a case of bi-.

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Vagus n Spinal accessory n Jugular foramen Jugular ganglion No dose ganglion Glassopharyngeal n Meningeal br Auricular br

(Muscles & mucosa of pharnyx, soft palate) Pharyngeal branch (Cricothyroid m) Superior laryngeal br

(Mucosa & glands of larnyx & epiplottis)

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